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Thanks to whomever suggest B2. I can really feel it working. Sorry, but people should know, it helps with my pooping.


Lisa says


10 years ago I fell very ill and was in hospital for a week. The consultant said they did not know what was wrong but that my white blood cell count was ‘way too high’. I was exhausted, the glands in my neck were so enlarged that I had lumps the size of oranges leaving me looking deformed (they carried out an ultrasound to look for non-hodgkins / hodgkins in the lumps but this was clear), my tonsils were swollen too. I was ill for 2 years following this suffering with weight loss, hundreds of mouth ulcers, UTI’s etc. I had further blood tests but the results went missing for 6 months so I assumed all clear when I didn’t hear anything then my doctor called saying it showed that I was vitamin B12 deficient but as it was 6 months ago and the ulcers had gone he would ‘file his papers’.

I’m 33 have a healthy diet but it’s been a running joke that I catch every bug going around.

Almost 2 years ago I caught Molluscum Contagiosum and have been unable to fight it off leaving me feeling very down.

My doctor finally carried out a blood test a week ago and I got a call 1 day later saying I need to see the doctor asap as I have a vitamin B12 deficiency.

I am seeing the doctor on Thursday but want to be more informed this time. Is it possible that I have been deficient all of this time? Could this explain why I seem to catch everything and not be able to fight it off like a normal healthy adult? I do have other symptoms (out of breath, palpitations tiredness).

Can this affect the immune system?

Any advice would be appreciated seen as my previous doctor just ‘filed his papers’!!

Thank you


Hi Lisa, My sons had Molloscum, and it took a very long time to go away. What really helped was Grandpa’s Pine Tar Soap in the bodywash form, and dabbing then with alcohol pads. Once they started washing and dabbing, the molloscum cleared up in two to four weeks.

When you say healthy diet, what do you mean?

Had you had some illness before you started getting sick all the time? About 20 years ago, this woman on the bus kept coughing right in my face. A few days later, I became ill for three weeks. My old family doctor, ” I don’t know what you got, but you got it good!” ( I miss my old doctor). After that, someone would have the sniffles, and I would get the flu. After I started taking my vitamins that I have listed elsewhere here, I did start having more resistance. I don’t exercise because of my asthma, but now that it’s cooler, I’ll start going for walks, and I am sure that that will help my resistance even more.

So, my advice to you, is to take my list of vitamins, plus B2, and do calm exercise like walking, even in cold weather, and you should start seeing an improvement before you know it.

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Statistical analyses were coordinated across centres by the Lipoprotein Investigators Collaborative (LIC). TRIUMPH analyses were conducted with SAS version 9.2 (SAS Institute, Cary, NC, USA) and R version 2.7.2 (R Foundation for Statistical Computing, Vienna, Austria); IHCS analyses were conducted with SPSS version 15.0 (IBM, Chicago, IL, USA) and SAS version 9.3 (SAS Institute, Cary, NC, USA). Two-tailed P -values <0.05 were considered statistically significant.

The baseline sociodemographic and clinical characteristics of the TRIUMPH and IHCS cohorts are shown in Table 1 . Patients were middle-aged to elderly, and more than two-thirds were men. Approximately, two-thirds of patients were white in TRIUMPH, whereas IHCS was predominantly white. About one-third of TRIUMPH patients and 20% of IHCS patients had diabetes mellitus. A minority of patients in both cohorts had heart failure. A discharge statin prescription was given to 87.5% in TRIUMPH, and nearly half of IHCS patients were discharged on a statin. There were relatively low rates of use of other lipid-modifying agents such as ezetimibe, fibrates, or niacin.

Baseline lipid profile and HDL-C subclass information are presented in Table 2 . Mean baseline levels of total cholesterol, LDL-C, non-HDL-C, and triglycerides were below goal levels for high-risk patients as defined by NCEP ATP III. HDL-C levels were low (TRIUMPH: 40 ± 10.6 mg/dL; IHCS: 34.6 ± 10.1 mg/dL), and the ratio of total cholesterol to HDL-C was high (TRIUMPH: 4.1 ± 1.2; IHCS: 4.5 ± 2.0). HDL 3 -C accounted for more than three-fourths of HDL-C; the mean ratio of HDL 3 -C to HDL-C was 0.78 ± 0.05 in both cohorts. Apolipoprotein A1 was more tightly correlated with HDL 3 -C than HDL 2 -C ( Supplementary material online, Table S1 ).


Baseline lipid parameters in TRIUMPH and IHCS

Mean (SD) or median (25th–75th percentile).

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Baseline lipid parameters in TRIUMPH and IHCS

Mean (SD) or median (25th–75th percentile).

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Follow-up was 100% complete in both studies at 2 years. In addition, in IHCS, follow-up was 89 and 46% complete at 3 and 5 years, respectively. Mortality occurred in 226 (9.2%) TRIUMPH and 243 (10.1%) IHCS patients. A total of 208 (8.6%) IHCS patients were rehospitalized for MI. There were no significant differences in mortality rates between tertiles of HDL-C ( Polo Shirt for Men Navy Blue Cotton 2017 L M S XL XXL Antony Morato Inexpensive Online 0ASWgy3px
). Within subclasses, there were also no differences between HDL 2 -C tertiles. However, for the lowest tertile of HDL 3 -C compared with the middle and highest tertiles, approximately two-fold higher rates of events were observed for mortality in TRIUMPH and mortality or MI in IHCS. This finding emerged by 1 year in IHCS, and remained consistent over time.

Study population In a previous study that established that N. gonorrhoeae, C. trachomatis and HSV could be causes of MPC, 779 randomly selected women aged 16–45 years who attended the Harborview Sexually Transmitted Disease (STD) Clinic between 1984 and 1986 for a new problem provided informed consent for microbiologic tests of cervical and vaginal specimens [ 6 , 7 ]. For the present analysis, samples of cervical secretions from 724 women remained, and specimens from 719 of the women proved to be amplifiable by PCR for purposes of M. genitalium detection. These 719 women were similar to the 60 women for whom samples were not available or not amplifiable with respect to demographic characteristics and MPC rates. However, women for whom samples were not available were more likely to have had chlamydial infection (32.7% of women whose samples were not available vs. 11.4% of women whose samples were) or bacterial vaginosis (BV) (49.1% vs. 32.2%). This difference was probably due to depletion of specimens that tested positive for these infections through use in previous studies

Study population

Clinical assessment A routine medical history was taken and recorded on a standardized form by a single experienced clinician (C.E.S.), who also performed external genital and bimanual pelvic examinations, as described elsewhere [ 6 , 7 ]. Samples obtained for microbiologic studies included cervical secretions collected on filter paper (Whatman #5) and stored at −20°C in 500 μL of PBS. “Mucopurulent cervicitis” was defined as the presence of either visible yellow mucopus or of ⩾30 polymorphonuclear leukocytes (PMNL)/1000× microscopic field on a Gram-stained smear of cervical mucus [ 7 ]. MPC was present in 215 (29.9%) of 719 women

Clinical assessment

Laboratory methods and detection of M. genitalium. N. gonorrhoeae, C. trachomatis HSV, M. hominis, Ureaplasma urealyticum , and Gardnerella vaginalis were detected by culture; T. vaginalis and Candida species, by culture and wet-mount microscopy; and BV, defined by Amsel’s criteria [ 23 ], as described elsewhere [ 7 ]. In the present study, cervical secretion specimens were rehydrated with 500 μL of 2SP (0.2 M sucrose, 0.02 M potassium phosphate buffer, and 0.001% phenol red [pH 7.5]), to compensate for desiccation during extended freezer storage, and processed by the Amplicor CT/NG specimen preparation kit (Roche Diagnostic Systems), according to the manufacturer’s directions (the “swab procedure”). This consisted of dilution of 50 μL of the patient specimen with an equal volume of transport medium, incubation for 10 min, dilution with 100 μL of sample diluent, and use of 50 μL of this processed specimen (12.5 μL of the original pretreated specimen) in the PCR. A similar amount of original pretreated specimen (16 μL) was used in PCRs after treatment to remove inhibitors (see below). To assess the suitability of the specimens for PCR after long-term freezer storage, we tested a subset of 40 specimens (20 from N. gonorrhoeae –positive women and 20 from N. gonorrhoeae –negative women, as previously determined by culture [ 7 ]) for N. gonorrhoeae by PCR (COBAS Amplicor; Roche Diagnostic Systems). We observed 100% concordance between these 2 tests, which indicated that bacterial DNA could be accurately detected in these specimens and that the specimens were suitable for PCR analysis

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